In the article, several studies indicate a potential correlation between air pollution exposure and Parkinson’s disease (PD). This research explores the results of traffic-related and multiple sources of particulate air pollution on PD in central California. Results showed that long-term exposure to local traffic-related air pollution (CO) and fine particulates from various sources (PM2.5) at homes and workplaces in central California correlated with an increased risk of PD. The impact of air pollution on the brain, particularly PD, prevalence, and risk, remains underexplored despite its recognition as a significant public health concern. While plausible mechanisms connect air pollutants to PD, epidemiological studies have produced inconsistent results, primarily due to long-term exposure assessment and outcome evaluation challenges. The study aimed to address these challenges by estimating the effect of long-term exposure to CO and PM2.5 on PD risk in a population-based case-control study conducted in central California, a region known for persistently high air pollution levels.

  This study relates to environmental science because it examines the relationship between air pollution, which is a primary environmental concern, and the risk of Parkinson’s disease (PD). The study clarifies the environmental elements contributing to neurodegenerative diseases by examining the long-term consequences of traffic-related pollution. The results highlight how important it is to comprehend how environmental exposures, such as CO and PM2.5, affect human health. Furthermore, the study explains the connection between neurological illnesses and environmental contamination, this way contributing to environmental science and public health. Ultimately, it emphasizes how urgent we need to take environmental initiatives to decrease the harmful consequences of air pollution on the population’s health.